La dynastie des Calvani : 3 romans (Volume multi thématique) (French Edition)

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The group II intron maturase: a reverse transcriptase and splicing factor go hand in hand. The splicing of group II introns in vivo requires the assistance of a multifunctional intron encoded protein IEP, or maturase. Each IEP is also a reverse-transcriptase enzyme that enables group II introns to behave as mobile genetic elements.

During splicing or retro-transposition, each group II intron forms a tight, specific complex with its own encoded IEP, resulting in a highly reactive holoenzyme. These structures explain how the same IEP scaffold is utilized for intron recognition, splicing and reverse transcription, while providing a physical basis for understanding the evolutionary transformation of the IEP into the eukaryotic splicing factor Prp8.

Detection of Copper II and Cadmium II binding to dissolved organic matter from macrophyte decomposition by fluorescence excitation-emission matrix spectra combined with parallel factor analysis. Both the protein-like and the humic-like components showed a marked quenching effect by Cu II. Negligible quenching effects were found for Cd II by components 1, 5 and 6.

The stability constants and the fraction of the binding fluorophores for humic-like components and Cu II can be influenced by macrophyte decomposition of various weight gradients in aquatic plants. Macrophyte decomposition within the scope of the appropriate aquatic phytomass can maximize the stability constant of DOM-metal complexes.

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A large amount of organic matter was introduced into the aquatic environment by macrophyte decomposition, suggesting that the potential risk of DOM as a carrier of heavy metal contamination in macrophytic lakes should not be ignored. The transcription factor ETS-1 regulates angiotensin II -stimulated fibronectin production in mesangial cells. Angiotensin II ANG II produced as result of activation of the renin-angiotensin system RAS plays a critical role in the pathogenesis of chronic kidney disease via its hemodynamic effects on the renal microcirculation as well as by its nonhemodynamic actions including the production of extracellular matrix proteins such as fibronectin, a multifunctional extracellular matrix protein that plays a major role in cell adhesion and migration as well as in the development of glomerulosclerosis.

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ETS-1 is an important transcription factor essential for normal kidney development and glomerular integrity. In addition, we characterized the role of ETS-1 on the transcriptional activation of fibronectin production in mesangial cells. We determined that ETS-1 directly activates the fibronectin promoter and by utilizing gel shift assays and chromatin immunoprecipitation assays identified two different ETS-1 binding sites that promote the transcriptional activation of fibronectin in response to ANG II.

In addition, we identified the essential role of CREB and its coactivator p on the transcriptional activation of fibronectin by ETS These studies unveil novel mechanisms involved in RAS-induced production of the extracellular matrix protein fibronectin in mesangial cells and establish the role of the transcription factor ETS-1 as a direct mediator of these effects.

The binding subunits of the insulin and insulin-like growth factor -I IGF I receptors from rat brain are of lower molecular weight than the corresponding receptor in rat liver, possibly due to variations in sialic acid content. The brain receptor is of smaller apparent mol wt about 10 K on sodium dodecyl sulfate polyacrylamide gel electrophoresis. This size difference is independent of ligand binding as it persists in iodinated and specifically immunoprecipitated receptors.

From studies of wheat germ agglutinin binding and the effect of neuraminidase on receptor mobility, we conclude that this difference is not simply due to variations in sialic acid content. Treatment with endoglycosidase F results in reduction in the molecular size of both liver and brain receptors and after this treatment the aglycoreceptors are of similar size. We conclude that in rat brain tissue the IGF II receptor like the binding subunits of the insulin and IGF I receptors is of lower molecular size than the corresponding receptors in rat liver. This difference is due to differences in N-linked glycosylation.

Full Text Available Introduction:Diabetes is a significant and expensive health problem which had influenced all the ages in almost all the countries.


Increasing prevalence of this disease has been caused by continued changes in lifestyle such as unsuitable nutrition, lack of physical activities and fatness which is often related to modern city life, mechanization and industrialization. It is an expensive disease, both for patients and the health and hygienic care systems. This research tried to examine the relationship between lifestyle risk factors and type II diabetes.

Methods: This research was a case-control type by random sampling and studying diabetes type II patients as case group and healthy people accompanying some other patients as the control group at Tonekabon Shahid Rajaei hospital.

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  • People were of both sexes, between 30 and 64 years of age and Tonekabon residents. The questionnaire used included demographic, nutritional, physical activities, stress tolerance and smoking status information. The SPSS Results: By analyzing the data collected, there was a meaningful statistical relationship between physical activities, stress residence, nutrition, smoking and the diabetes type II disease P-value0.

    Conclusion: Results imply that some risk factors important in diabetes type II include unsuitable nutrition such as having too much of sweets and sugar,lack of fruits, vegetables, fish, proteins and also lack of physical activities, stress tolerance and control. Its expression in humans is tightly controlled by maternal imprinting, a genetic restraint that is lost in many cancers, resulting in up-regulation of both mature IGF- II mRNA and protein expression. To date, it is ambiguous as to what role these IGF- II isoforms have in initiating and sustaining tumorigenesis and whether they are bioavailable.

    We have expressed each individual IGF- II isoform in their proper O-glycosylated format and established that all bind to the IGF-I receptor and both insulin receptors A and B, resulting in their activation and subsequent stimulation of fibroblast proliferation. IGF- II isoforms thus possess unique biological properties that may enable them to escape normal sequestration avenues and remain bioavailable in vivo to sustain oncogenic signaling.

    Greenall, Sameer A.


    A family of insulin-like growth factor II mRNA-binding proteins represses translation in late development. Mouse IMPs are produced in a burst at embryonic day In human vaccination, the response to PA can vary greatly, and the response is often directed toward non-neutralizing epitopes. Variable vaccine responses have been shown to be due in part to genetic differences in individuals, with both MHC class II and other genes playing roles.

    H2k H-2k. Common epitopes, reactive in all strains, occurred in both LF and PA responses. A temperature-dependent theory for He II : Application to the liquid structure factor. A temperature-dependent theory is presented for He II , which is based on both a gauge-theoretic formulation as well as a mean-field Hartree approach. A simple model calculation is then performed within this framework for the liquid structure factor of the system. In particular, explicit expressions are obtained for the low-momentum-transfer and low-temperature limits, which seem to conform with the available experimental data.

    Further, the curvature of the structure factor is predicted, under these circumstances, to be only mildly dependent on temperature. Throughout, we compare and contrast with other theoretical attempts, including Feynman's. Insulin-like growth factor-II receptors in cultured rat hepatocytes: regulation by cell density. Insulin-like growth factor-II IGF- II receptors in primary cultures of adult rat hepatocytes were characterized and their regulation by cell density examined. In hepatocytes cultured at 5 X 10 5 cells per 3. IGF- II binding increased when cells were permeabilized with 0. Determined by Scatchard analysis and by polyacrylamide gel electrophoresis after affinity labeling, the higher binding was due solely to an increase in binding sites present on kDa type II IGF receptors.

    The increase was not due simply to the exposure of receptors from the intracellular pool, as a density-related increase in receptors was also seen in cells permeabilized with digitonin. There was no evidence that IGF binding proteins, either secreted by hepatocytes or present in fetal calf serum, had any effect on the measurement of receptor concentration or affinity. We conclude that rat hepatocytes in primary culture contain specific IGF- II receptors and that both cell surface and intracellular receptors are regulated by cell density.

    Significance of determination of the serum levels of homocysteine Hcy and insulin-like growth factor-II IGF- II in patients with cerebrovascular diseases.

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    H19 RNA is a major oncofetal 2. The maternally expressed H19 gene is located 90 kilobase pairs downstream from the paternally expressed insulin-like growth factor II IGF- II gene on human chromosome 11 and mouse chromosome 7; and due to their recip The maternally expressed H19 gene is located 90 kilobase pairs downstream from the paternally expressed insulin-like growth factor II IGF- II gene on human chromosome 11 and mouse chromosome 7; and due La gestion de las aguas subterraneas en el acuifero Mancha Occidental.

    Los resultados obtenidos pe Reflexiones sobre el silencio y el lenguaje a la luz de oriente y occidente. Nuestra experiencia del mundo y de nosotros mismos es This study aimed to investigate risk factors accompanied with nephropathy in patients with type II diabetes based on the biopsychosocial model.

    The data were collected by demographical information questionnaire along with disease characteristics and depression anxiety stress scales dass, quality of life scale who- qol- bref, diabetes self-management scale dsms, and diabetes knowledge scale dks, then analyzed by chi-square, independent t-test and logistic regression with pasw software.

    Conclusion: It is important to pay attention to hypertension, glycated hemoglobin, body mass index, diabetes self-management, depression, quality of life, and diabetes knowledge for therapeutic intervention programming and diabetes complications control protocols for diabetic patients. Risk factors for periodontal diseases among Yemeni type II diabetic patients.

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    A case-control study. Full Text Available Background: Chronic periodontal diseases are one of diabetes mellitus complications. The present study aims to compare the periodontal status of type II diabetic patients to a control group and assess the role of risk factors in both groups. Materials and methods: A case-control study was conducted of individuals type II diabetics and non-diabetics. Full mouth periodontal examination including plaque index, gingival bleeding, gingival recession, clinical attachment loss CAL, tooth mobility, furcation involvement and the number of missing teeth.

    The diabetic group was also subdivided according to smoking and Khat chewing habits. Result: The severity of periodontal disease among type II diabetic patients were significantly higher compared to the control group regarding the plaque index 2. Conclusion: Type II diabetic patients have severe periodontal destruction and tooth loss compared to non-diabetic people and there were no differences within the diabetic group in regards to smoking and Khat chewing habits.

    Angiotensin II upregulates the expression of placental growth factor in human vascular endothelial cells and smooth muscle cells.

    Full Text Available Abstract Background Atherosclerosis is now recognized as a chronic inflammatory disease. Angiotensin II Ang II is a critical factor in inflammatory responses, which promotes the pathogenesis of atherosclerosis. Placental growth factor PlGF is a member of the vascular endothelial growth factor VEGF family cytokines and is associated with inflammatory progress of atherosclerosis.

    Nucleon form factors in dispersively improved chiral effective field theory. Electromagnetic form factors.